An Autopsy Case of Transthyretin-Related Cardiac Amyloidosis (TTR-CA), Which Fell into Low-Output Syndrome after Cardiac Surgery
Author:
Affiliation:
1. Department of Cardiovascular Surgery, Saiseikai Izuo Hospital
2. Department of Cardiovascular Surgery, Takanohara Central Hospital
Publisher
The Japanese Society for Cardiovascular Surgery
Subject
Psychiatry and Mental health
Link
https://www.jstage.jst.go.jp/article/jjcvs/51/3/51_157/_pdf
Reference28 articles.
1. 1) Gonzalez-Lopez E, Gallego-Delgado M, Guzzo-Merello G et al. Wild-type transthyretin amyloidosis as a cause of heart failure with preserved ejection fraction. Eur Heart J 2015 ; 36 : 2585-94.
2. 2) Mohammed SF, Mirzoyev SA, Edwards WD et al. Left ventricular amyloid deposition in patients with heart failure and preserved ejection fraction. LACC Heart Fail 2014 ; 2 : 113-22.
3. 3) Zhang KW, Stockerl-Goldstein KE, Lenihan DJ. Emerging therapeutics for the treatment of light chain and transthyretin amyloidosis. JACC Basic Transl Sci 2019 ; 4 : 438-48.
4. 4) Galant NJ, Westermark P, Higaki JN et al. Transthyretin amyloidosis : an under-recognized neuropathy and cardiomyopathy. Clin Sci 2017 ; 131 : 395-409.
5. 5) Kagan BL, Thundimadathil J. Amyloid peptide pores and the beta sheet conformation. Adv Exp Med Biol 2010 ; 677 : 150-67.
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