Mitochondrial Inhibitor Models of Huntington’s Disease and Parkinson’s Disease Induce Zinc Accumulation and Are Attenuated by Inhibition of Zinc Neurotoxicity in vitro or in vivo-Reference-Cited by-同舟云学术

Mitochondrial Inhibitor Models of Huntington’s Disease and Parkinson’s Disease Induce Zinc Accumulation and Are Attenuated by Inhibition of Zinc Neurotoxicity in vitro or in vivo

Published:2012-05-24 Issue:1 Volume:11 Page:49-58
ISSN:1660-2854
Container-title:Neurodegenerative Diseases
language:en
Short-container-title:Neurodegener Dis

Author:

Sheline Christian T.,Zhu Julia,Zhang Wendy,Shi Chunxiao,Cai Ai-Li

Publisher

S. Karger AG

Subject

Neurology (clinical),Neurology

Reference49 articles.

1. Energetics in the pathogenesis of neurodegenerative diseases

2. Recent advances in understanding the pathogenesis of Huntington's disease

3. Oxidative damage and metabolic dysfunction in Huntington's disease: Selective vulnerability of the basal ganglia

4. Neurochemical and histologic characterization of striatal excitotoxic lesions produced by the mitochondrial toxin 3-nitropropionic acid

5. 3-Nitropropionic Acid - Exogenous Animal Neurotoxin and Possible Human Striatal Toxin

Cited by 42 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Monoterpenoid indole alkaloid adducts and dimers from Melodinus fusiformis;Phytochemistry;2023-07

2. Dyshomeostasis of Iron and Its Transporter Proteins in Cypermethrin-Induced Parkinson’s Disease;Molecular Neurobiology;2023-06-23

3. Zinc and Central Nervous System Disorders;Nutrients;2023-04-29

4. Zinc release from mitochondria contributes to MPP+-induced lysosomal disruption and neuronal death;2023-01-18

5. The role of d-block metal ions in neurodegenerative diseases;Comprehensive Inorganic Chemistry III;2023