Aldehyde Dehydrogenase, Aldose Reductase, and Free Radical Scavengers in Cataract
Author:
Publisher
S. Karger AG
Subject
Biochemistry
Cited by 16 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Telomere-dependent senescent phenotype of lens epithelial cells as a biological marker of aging and cataractogenesis: the role of oxidative stress intensity and specific mechanism of phospholipid hydroperoxide toxicity in lens and aqueous;Fundamental & Clinical Pharmacology;2011-03-09
2. Mitochondria induce oxidative stress, generation of reactive oxygen species and redox state unbalance of the eye lens leading to human cataract formation: disruption of redox lens organization by phospholipid hydroperoxides as a common basis for cataract;Cell Biochemistry and Function;2011-03-07
3. Synthesis and biological evaluations of novel bendazac lysine analogues as potent anticataract agents;Bioorganic & Medicinal Chemistry Letters;2010-04
4. Molecular Cloning and Oxidative Modification of Human Lens ALDH1A1: Implication in Impaired Detoxification of Lipid Aldehydes;Journal of Toxicology and Environmental Health, Part A;2009-03-27
5. Role of Aldehyde Dehydrogenase Isozymes in the Defense of Rat Lens and Human Lens Epithelial Cells against Oxidative Stress;Investigative Opthalmology & Visual Science;2005-01-01
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