Overexpression of Dyrk1A Causes the Defects in Synaptic Vesicle Endocytosis
Author:
Publisher
S. Karger AG
Subject
Cellular and Molecular Neuroscience,Developmental Neuroscience,Neurology
Reference39 articles.
1. Down syndrome phenotypes: the consequences of chromosomal imbalance.
2. Overexpression of Dyrk1A contributes to neurofibrillary degeneration in Down syndrome
3. DYRK1A BAC transgenic mice show altered synaptic plasticity with learning and memory defects
4. Microdeletion of the Down syndrome critical region at 21q22
5. DYRK1A accumulates in splicing speckles through a novel targeting signal and induces speckle disassembly
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