New Evidence of Neuroprotection by Lactate after Transient Focal Cerebral Ischaemia: Extended Benefit after Intracerebroventricular Injection and Efficacy of Intravenous Administration-Reference-Cited by-同舟云学术

New Evidence of Neuroprotection by Lactate after Transient Focal Cerebral Ischaemia: Extended Benefit after Intracerebroventricular Injection and Efficacy of Intravenous Administration

Published:2012 Issue:5-6 Volume:34 Page:329-335
ISSN:1015-9770
Container-title:Cerebrovascular Diseases
language:en
Short-container-title:Cerebrovasc Dis

Author:

Berthet Carole,Castillo Ximena,Magistretti Pierre J.,Hirt Lorenz

Publisher

S. Karger AG

Subject

Cardiology and Cardiovascular Medicine,Clinical Neurology,Neurology

Reference22 articles.

1. Glutamate uptake into astrocytes stimulates aerobic glycolysis: a mechanism coupling neuronal activity to glucose utilization.

2. Astrocyte-Neuron Lactate Transport Is Required for Long-Term Memory Formation

3. Brain Lactate Is an Obligatory Aerobic Energy Substrate for Functional Recovery After Hypoxia: Further In Vitro Validation

4. Brain lactate, not glucose, fuels the recovery of synaptic function from hypoxia upon reoxygenation: an in vitro study

5. Lactate reduces glutamate-induced neurotoxicity in rat cortex

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