Expression and Effects of IL-33 and ST2 in Allergic Bronchial Asthma: IL-33 Induces Eotaxin Production in Lung Fibroblasts-Reference-Cited by-同舟云学术

Expression and Effects of IL-33 and ST2 in Allergic Bronchial Asthma: IL-33 Induces Eotaxin Production in Lung Fibroblasts

Published:2011 Issue:s1 Volume:155 Page:12-20
ISSN:1423-0097
Container-title:International Archives of Allergy and Immunology
language:en
Short-container-title:Int Arch Allergy Immunol

Author:

Kurokawa Masatsugu,Matsukura Satoshi,Kawaguchi Mio,Ieki Koushi,Suzuki Shintaro,Odaka Miho,Watanabe Shin,Homma Tetsuya,Sato Masayuki,Yamaguchi Munehiro,Takeuchi Hiroko,Adachi Mitsuru

Publisher

S. Karger AG

Subject

Immunology,General Medicine,Immunology and Allergy

Reference41 articles.

1. A putative protein of a growth specific cDNA from BALB/C-3T3 cells is highly similar to the extracellular portion of mouse interleukin 1 receptor

2. Presence of a novel primary response gene ST2L, encoding a product highly similar to the interleukin 1 receptor type 1

3. IL-33, an Interleukin-1-like Cytokine that Signals via the IL-1 Receptor-Related Protein ST2 and Induces T Helper Type 2-Associated Cytokines

4. Cutting Edge: The ST2 Ligand IL-33 Potently Activates and Drives Maturation of Human Mast Cells

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