Involvement of SSAO/VAP-1 in Oxygen-Glucose Deprivation-Mediated Damage Using the Endothelial hSSAO/VAP-1-Expressing Cells as an Experimental Model of Cerebral Ischemia
Author:
Publisher
S. Karger AG
Subject
Cardiology and Cardiovascular Medicine,Neurology (clinical),Neurology
Cited by 17 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Acute high‐altitude hypoxia exposure causes neurological deficits via formaldehyde accumulation;CNS Neuroscience & Therapeutics;2022-05-18
2. SSAO/VAP-1 in Cerebrovascular Disorders: A Potential Therapeutic Target for Stroke and Alzheimer’s Disease;International Journal of Molecular Sciences;2021-03-25
3. Endothelium-Targeted Deletion of microRNA-15a/16-1 Promotes Poststroke Angiogenesis and Improves Long-Term Neurological Recovery;Circulation Research;2020-04-10
4. Blood-brain barrier dysfunction underlying Alzheimer's disease is induced by an SSAO/VAP-1-dependent cerebrovascular activation with enhanced Aβ deposition;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2019-09
5. Reactive species-induced microvascular dysfunction in ischemia/reperfusion;Free Radical Biology and Medicine;2019-05
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