Could a Possible Crosstalk between AMPK and TGF-β Signaling Pathways Be a Key Player in Benign and Malignant Salivary Gland Tumors?-Reference-Cited by-同舟云学术

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Could a Possible Crosstalk between AMPK and TGF-β Signaling Pathways Be a Key Player in Benign and Malignant Salivary Gland Tumors?

Published:2012 Issue:12 Volume:35 Page:770-774
ISSN:2296-5270
Container-title:Oncology Research and Treatment
language:en
Short-container-title:Oncol Res Treat

Author:

Ghahhari Nastaran M.,Ghahhari Hamed M.,Kadivar Mehdi

Publisher

S. Karger AG

Subject

Cancer Research,Oncology,Hematology

Reference34 articles.

1. Establishment and characterization of pleomorphic adenoma cell systems: an in-vitro demonstration of carcinomas arising secondarily from adenomas in the salivary gland

2. Fusion oncogenes and tumor type specificity—insights from salivary gland tumors

3. Apoptosis and apoptotic-related factors in mucoepidermoid carcinoma of the oral minor salivary glands

Cited by 6 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. TGFβ signaling pathway in salivary gland tumors;Archives of Oral Biology;2024-06

2. The Molecular Biology of Benign and Malignant Salivary Gland Tumors;Salivary Gland Pathology;2021-07-23

3. Negative regulation of TGF-β by AMPK and implications in the treatment of associated disorders;Acta Biochimica et Biophysica Sinica;2018-03-22

4. Inactivation of the LKB1-AMPK signaling pathway does not contribute to salivary gland tumor development - a short report;Cellular Oncology;2016-08

5. Role of Smad signaling in kidney disease;International Urology and Nephrology;2015-10-03

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