Prokr2-Deficient Mice Display Vascular Dysmorphology of the Fetal Testes: Potential Implications for Kallmann Syndrome Aetiology
Author:
Publisher
S. Karger AG
Subject
Developmental Biology,Embryology,Endocrinology, Diabetes and Metabolism
Reference46 articles.
1. Loss-of-Function Mutations in the Genes Encoding Prokineticin-2 or Prokineticin Receptor-2 Cause Autosomal Recessive Kallmann Syndrome
2. Expression profiling of purified mouse gonadal somatic cells during the critical time window of sex determination reveals novel candidate genes for human sexual dysgenesis syndromes
3. KAL1 mutations are not a common cause of idiopathic hypogonadotrophic hypogonadism in humans
4. Using real time RT-PCR analysis to determine multiple gene expression patterns during XX and XY mouse fetal gonad development
5. Hypogonadisme hypogonadotrope : notions récentes sur la régulation de l’axe hypothalamo-hypophyso-gonadique
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1. Immune and vascular contributions to organogenesis of the testis and ovary;The FEBS Journal;2021-04-12
2. Prokineticin receptor 2 affects GnRH3 neuron ontogeny but not fertility in zebrafish;Scientific Reports;2020-05-06
3. Correlations Among Genotype and Outcome in Chinese Male Patients With Congenital Hypogonadotropic Hypogonadism Under HCG Treatment;The Journal of Sexual Medicine;2020-04
4. Clinical and genetic features of 64 young male paediatric patients with congenital hypogonadotropic hypogonadism;Clinical Endocrinology;2017-09-13
5. A heterozygous microdeletion of 20p12.2-3 encompassing PROKR2 and BMP2 in a patient with congenital hypopituitarism and growth hormone deficiency;American Journal of Medical Genetics Part A;2017-06-06
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