GLTSCR2 Sensitizes Cells to Hypoxic Injury without Involvement of Mitochondrial Apoptotic Cascades-Reference-Cited by-同舟云学术

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GLTSCR2 Sensitizes Cells to Hypoxic Injury without Involvement of Mitochondrial Apoptotic Cascades

Published:2007 Issue:5 Volume:74 Page:301-308
ISSN:1015-2008
Container-title:Pathobiology
language:en
Short-container-title:Pathobiology

Author:

Yim Ji-Hye,Kim Yong-Jun,Cho Young-Eun,Ko Jeong-Hun,Kim Su-Mi,Kim Jee-Youn,Park Jae-Hoon

Publisher

S. Karger AG

Subject

Cell Biology,Molecular Biology,General Medicine,Pathology and Forensic Medicine

Reference26 articles.

1. HIF-1α and p53 Promote Hypoxia-Induced Delayed Neuronal Death in Models of CNS Ischemia

2. Oxygen sensing and molecular adaptation to hypoxia

3. Mechanisms of cell death in hypoxia/reoxygenation injury

4. Expression of the gene encoding the proapoptotic Nip3 protein is induced by hypoxia

5. The role of hypoxia inducible factor 1 (HIF-1) in hypoxia induced apoptosis

Cited by 6 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The pre‐rRNA processing factor Nop53 regulates fungal development and pathogenesis via mediating production of reactive oxygen species;Environmental Microbiology;2018-03-26

2. PICT-1 is a key nucleolar sensor in DNA damage response signaling that regulates apoptosis through the RPL11-MDM2-p53 pathway;Oncotarget;2016-11-04

3. The expression of GLTSCR2 in cervical intra-epithelial lesion and cancer;Archives of Gynecology and Obstetrics;2014-08-14

4. GLTSCR2 contributes to the death resistance and invasiveness of hypoxia-selected cancer cells;FEBS Letters;2012-07-28

5. Post-Translational Modifications of PTEN and their Potential Therapeutic Implications;Current Cancer Drug Targets;2011-06-01

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