Loss of Heterozygosity Status of D9S105 Marker Is Associated with Downregulation of Krüppel-Like Factor 4 Expression in Pancreatic Ductal Adenocarcinoma and Pancreatic Intraepithelial Lesions-Reference-Cited by-同舟云学术

Loss of Heterozygosity Status of D9S105 Marker Is Associated with Downregulation of Krüppel-Like Factor 4 Expression in Pancreatic Ductal Adenocarcinoma and Pancreatic Intraepithelial Lesions

Published:2011-06 Issue:1 Volume:11 Page:30-42
ISSN:1424-3903
Container-title:Pancreatology
language:en
Short-container-title:Pancreatology

Author:

Funel Niccola,Morelli Mariangela,Giovannetti Elisa,Del Chiaro Marco,Pollina Luca Emanuele,Mosca Franco,Boggi Ugo,Cavazzana Andrea,Campani Daniela

Publisher

Elsevier BV

Subject

Gastroenterology,Hepatology,Endocrinology, Diabetes and Metabolism

Reference43 articles.

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4. Role of gut-enriched Krüppel-like factor in colonic cell growth and differentiation;Shie;Am J Physioi Gastrointest Liver Physiol,2000

5. Transactivation and growth suppression by the gut-enriched Krüppel-like factor (Krüppel-like factor 4) are dependent on acidic amino acid residues and protein-protein interaction;Geiman;Nucleic Acids Res,2000

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2. Prognostic significance of KLF4 in solid tumours: an updated meta-analysis;BMC Cancer;2022-02-17

3. Krüppel-like factor 4 expression in solid tumor prognosis: A meta-analysis;Clinica Chimica Acta;2018-10

4. Novel crosstalk between KLF4 and ZEB1 regulates gemcitabine resistance in pancreatic ductal adenocarcinoma;International Journal of Oncology;2017-08-22

5. DNA-Methyltransferase 1 Induces Dedifferentiation of Pancreatic Cancer Cells through Silencing of Krüppel-Like Factor 4 Expression;Clinical Cancer Research;2017-06-28