Prevention of Hypertension with or without Renin-Angiotensin System Inhibition Precludes Nephrin Loss in the Early Stage of Experimental Diabetes Mellitus-Reference-Cited by-同舟云学术

Prevention of Hypertension with or without Renin-Angiotensin System Inhibition Precludes Nephrin Loss in the Early Stage of Experimental Diabetes Mellitus

Published:2007 Issue:2 Volume:107 Page:p57-p64
ISSN:1660-2137
Container-title:Nephron Physiology
language:en
Short-container-title:Nephron Physiol

Author:

Amazonas Roberto Bleuel,de Almeida Sanita Rodrigo,Kawachi Hiroshi,Lopes de Faria José B.

Publisher

S. Karger AG

Subject

Physiology (medical),Nephrology,General Medicine,Physiology

Reference33 articles.

1. Systolic blood pressure relates to the rate of progression of albuminuria in NIDDM

2. Association of systolic blood pressure with macrovascular and microvascular complications of type 2 diabetes (UKPDS 36): prospective observational study

3. Blood Pressure Lowering for the Prevention and Treatment of Diabetic Kidney Disease

4. Effect of captopril on progression to clinical proteinuria in patients with insulin-dependent diabetes mellitus and microalbuminuria. European Microalbuminuria Captopril Study Group

5. Effective Antihypertensive Treatment Postpones Renal Insufficiency in Diabetic Nephropathy

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1. ASSESSMENT OF NEPHRIN AND PODOCIN LEVELS IN THE URINE OF PATIENTS WITH DIABETES MELLITUS;Nephrology (Saint-Petersburg);2017-03-03

2. Specific Mechanisms Underlying Right Heart Failure: The Missing Upregulation of Superoxide Dismutase-2 and Its Decisive Role in Antioxidative Defense;Antioxidants & Redox Signaling;2015-11-20

3. Urinary excretion of markers for podocyte injury in patients with diabetes mellitus;Terapevticheskii arkhiv;2015

4. The contribution of hypertension to diabetic nephropathy and retinopathy: the role of inflammation and oxidative stress;Hypertension Research;2011-01-13

5. Angiotensin-(1–7) reduces proteinuria and diminishes structural damage in renal tissue of stroke-prone spontaneously hypertensive rats;American Journal of Physiology-Renal Physiology;2011-01