Foreign Gene Transfer to Cardiomyocyte Using a Replication-Defective Recombinant Coxsackievirus B3 without Cytotoxicity
Author:
Publisher
S. Karger AG
Subject
Infectious Diseases,Virology
Reference20 articles.
1. Low-Level Expression of a Mutant Coxsackieviral cDNA Induces a Myocytopathic Effect in Culture
2. Transgenic expression of replication-restricted enteroviral genomes in heart muscle induces defective excitation-contraction coupling and dilated cardiomyopathy.
3. Defective RNA replication by poliovirus mutants deficient in 2A protease cleavage activity
4. Long-term cardiac gene expression using a coxsackieviral vector
5. Cardioselective Infection With Coxsackievirus B3 Requires Intact Type I Interferon Signaling
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1. Specific elimination of coxsackievirus B3 infected cells with a protein engineered toxin-antitoxin system;Molecular & Cellular Toxicology;2019-09-30
2. Arrayed CRISPR screen with image-based assay reliably uncovers host genes required for coxsackievirus infection;Genome Research;2018-04-30
3. Cholic Acid Attenuates ER Stress-Induced Cell Death in Coxsackievirus-B3 Infection;Journal of Microbiology and Biotechnology;2018-01-28
4. Fructus Amomi Cardamomi Extract Inhibits Coxsackievirus-B3 Induced Myocarditis in a Murine Myocarditis Model;Journal of Microbiology and Biotechnology;2016-11-28
5. Soluble Coxsackievirus B3 3C Protease Inhibitor Prevents Cardiomyopathy in an Experimental Chronic Myocarditis Murine Model;Virus Research;2015-03
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