Renal Sodium-Glucose Cotransporter Activity and Aquaporin-2 Expression in Rat Kidney during Chronic Nitric Oxide Synthase Inhibition-Reference-Cited by-同舟云学术

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Renal Sodium-Glucose Cotransporter Activity and Aquaporin-2 Expression in Rat Kidney during Chronic Nitric Oxide Synthase Inhibition

Published:2007 Issue:3 Volume:107 Page:p77-p86
ISSN:1660-2137
Container-title:Nephron Physiology
language:en
Short-container-title:Nephron Physiol

Author:

Albertoni Borghese María F.,Majowicz Mónica P.,Ortiz María C.,Delgado María F.,Sterin Speziale Norma B.,Vidal Noberto A.

Publisher

S. Karger AG

Subject

Physiology (medical),Nephrology,General Medicine,Physiology

Reference48 articles.

1. Chronic inhibition of nitric oxide synthesis. A new model of arterial hypertension.

2. Evolution of Chronic Nitric Oxide Inhibition Hypertension

3. UPREGULATION OF VASCULAR RENIN–ANGIOTENSIN AND ENDOTHELIN SYSTEMS IN RATS INHIBITED OF NITRIC OXIDE SYNTHESIS

4. Evidence that the Autonomic Nervous System Plays a Major Role in the L-NAME - Induced Hypertension in Conscious Rats

5. Mechanism of vasoconstriction induced by chronic inhibition of nitric oxide in rats.

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1. Altered expression of Aquaporin-2 in one-kidney, one-clip hypertension;Life Sciences;2018-09

2. Aquaporin Membrane Channels in Oxidative Stress, Cell Signaling, and Aging: Recent Advances and Research Trends;Oxidative Medicine and Cellular Longevity;2018

3. Aquaporins, Vasopressin, and Aging: Current Perspectives;Endocrinology;2015-03-01

4. SGLT2 inhibitors: their potential reduction in blood pressure;Journal of the American Society of Hypertension;2015-01

5. Renal Response to L-Arginine in Diabetic Rats. A Possible Link between Nitric Oxide System and Aquaporin-2;PLoS ONE;2014-08-11

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