Renal Lesions of Hyperlipidemic Imai Rats: A Spontaneous Animal Model of Focal Glomerulosclerosis
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S. Karger AG
Cited by 33 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Protective effects of Rho kinase inhibitor fasudil on rats with chronic kidney disease;American Journal of Physiology-Renal Physiology;2013-06-01
2. Participation of endoplasmic reticulum stress in the pathogenesis of spontaneous glomerulosclerosis–Role of intra-renal angiotensin system;Translational Research;2012-10
3. Role of Intrarenal Angiotensin System Activation, Oxidative Stress, Inflammation, and Impaired Nuclear Factor-Erythroid-2-Related Factor 2 Activity in the Progression of Focal Glomerulosclerosis;Journal of Pharmacology and Experimental Therapeutics;2011-02-25
4. Effects of pioglitazone and candesartan on renal fibrosis and the intrarenal plasmin cascade in spontaneously hypercholesterolemic rats;American Journal of Physiology-Renal Physiology;2007-10
5. Male Gender and Not the Severity of Hypertension Is Associated With End-Organ Damage in Aged Stroke-Prone Spontaneously Hypertensive Rats;American Journal of Hypertension;2005-06
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