Secondary Bacteremia After Rotavirus Gastroenteritis in Infancy

Author:

Lowenthal Alexander1,Livni Gilat123,Amir Jacob24,Samra Zmira5,Ashkenazi Shai123

Affiliation:

1. Departments of Pediatrics A

2. Unit of Infectious Diseases, Schneider Children’s Medical Center of Israel, Petach Tikva, Israel

3. Felsenstein Medical Research Center, Petach Tikva, Israel

4. C

5. Department of Clinical Microbiology, Rabin Medical Center, Petach Tikva, Israel

Abstract

We describe 1 neonate and 3 infants with bacteremia secondary to rotavirus gastroenteritis. All had a typical course of an increase in temperature several days after admission. The causative organisms were Enterobacter cloacae and Klebsiella pneumoniae, both normal commensals of the small intestine. Pediatricians should be aware of this complication.

Publisher

American Academy of Pediatrics (AAP)

Subject

Pediatrics, Perinatology and Child Health

Reference10 articles.

1. Parashar UD, Hummelman EG, Bresee JS, Miller MA, Glass RI. Global illness and deaths caused by rotavirus disease in children. Emerg Infect Dis. 2003;9:565–572

2. Lundgren O, Svensson L. Pathogenesis of rotavirus diarrhea. Microbes Infect. 2001;3:1145–1156

3. Ramig RF. Pathogenesis of intestinal and systemic rotavirus infection. J Virol. 2004;78:10213–10220

4. Drasar BS, Barrow PA. The bacteria flora of the normal intestine. In: Draser BS, Barrow PA, eds. Intestinal Microbiology. Washington, DC: American Society for Microbiology; 1985

5. National Committee for Clinical Laboratory Standards. Performance Standards for Antimicrobial Disk Susceptibility Tests for Bacteria That Grow Aerobically: Approved Standard M2-A7. Villanova, PA: The Committee; 2003

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