Both Relative Insulin Resistance and Defective Islet β-Cell Processing of Proinsulin Are Responsible for Transient Hyperglycemia in Extremely Preterm Infants
Author:
Affiliation:
1. Service de Réanimation Pédiatrique et Néonatale, Hôpital Necker-Enfants Malades, Paris, France
2. Laboratoire d’Hormonologie, Hôpital Saint Vincent de Paul, Paris, France
3. Service de Néonatologie, Institut de Puériculture, Paris, France
Abstract
Publisher
American Academy of Pediatrics (AAP)
Subject
Pediatrics, Perinatology and Child Health
Link
https://publications.aap.org/pediatrics/article-pdf/113/3/537/1014973/zpe00304000537.pdf
Reference23 articles.
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2. Hawdon JM, Aynsley-Green A, Alberti KG, Ward Platt MP. The role of pancreatic insulin secretion in neonatal glucoregulation. I. Healthy term and preterm infants. Arch Dis Child.1993;68:274–279
3. Tyrala EE, Chen X, Boden G. Glucose metabolism in the infant weighing less than 1100 grams. J Pediatr.1994;125:283–287
4. Pollak A, Cowett RM, Schwartz R, Oh W. Glucose disposal in low birth weight infants during steady state hyperglycemia: effects of exogenous insulin administration. Pediatrics.1978;61:546–549
5. Hawdon JM, Hubbard M, Hales CN, Clark PM. Use of specific immunoradiometric assay to determine preterm neonatal insulin-glucose relations. Arch Dis Child.1995;73:F166–F169
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