Author:
Ohno T.,Bando M.,Nagura H.,Ishii K.,Yamanouchi H.
Abstract
Article abstract The authors report a patient of pure apraxic agraphia with normal praxis due to left thalamic infarction. 15O-gas-PET showed reduced oxygen metabolism in the left thalamus and the left dorsolateral premotor area, while MRI and 11C-fulumazenil-PET showed no remarkable lesions in the frontal cortex. The patient’s word imaging remained normal. The authors hypothesize that thalamic destruction causes pure apraxic agraphia by exerting a remote effect on left dorsolateral premotor area and blocking somewhere between graphemic area and motor programming.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Cited by
29 articles.
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