Disappearance of Both the BCR/ABL1 Fusion Gene and the JAK2V617F Mutation with Dasatinib Therapy in a Patient with Imatinib-Resistant Chronic Myelogenous Leukemia
Author:
Publisher
Japanese Society for Lymphoreticular Tissue Research
Subject
General Medicine
Reference12 articles.
1. 1 Scott LM, Campbell PJ, Baxter EJ, Tony Todd T, Stephens P, et al.: The V617F JAK2 mutation is uncommon in cancers and in myeloid malignancies other than the classic myeloproliferative disorders. Blood 106:2920-2921, 2005
2. 2 Kramer A, Reiter A, Kruth J, Erben P, Hochhaus A, et al.: JAK2-V617F mutation in a patient with Philadelphia-chromosome-positive chronic myeloid leukaemia. Lancet Oncol 8:658-660, 2007
3. 3 Hussein K, Bock O, Seegers A, Flasshove M, Henneke F, et al.: Myelofibrosis evolving during imatinib treatment of a chronic myeloproliferative disease with coexisting BCR-ABL translocation and JAK2V617F mutation. Blood 109:4106-4107, 2007
4. 4 Conchon MR, Costa JL, Novaes MM, Dorlhiac-Llacer PE, de Alencar Fischer Chamone D, et al.: Simultaneous detection of JAK2 V617F mutation and bcr-abl translocation in a patient with chronic myelogenous leukemia. Int J Hematol 88:243-245, 2008
5. 5 Jallades L, Hayette S, Tigaud I, Johnston A, Coiffier B, et al.: Emergence of therapy-unrelated CML on a background of BCR-ABL-negative JAK2V617F-positive chronic idiopathic myelofibrosis. Leuk Res 32:1608-1610, 2008
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