Seizure as the main presenting manifestation of three patients with acute glufosinate-ammonium poisoning
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Publisher
Zhejiang University Press
Link
https://link.springer.com/content/pdf/10.1631/jzus.B2300469.pdf
Reference16 articles.
1. Cha YS, Kim H, Lee Y, et al., 2018. The relationship between serum ammonia level and neurologic complications in patients with acute glufosinate ammonium poisoning: a prospective observational study. Hum Exp Toxicol, 37(6): 571–579. https://doi.org/10.1177/0960327117715902
2. Hermenegildo C, Monfort P, Felipo V, 2000. Activation of N-methyl-d-aspartate receptors in rat brain in vivo following acute ammonia intoxication: characterization by in vivo brain microdialysis. Hepatology, 31(3):709–715. https://doi.org/10.1002/hep.510310322
3. Hoerlein G, 1994. Glufosinate (phosphinothricin), a natural amino acid with unexpected herbicidal properties. In: Ware GW (Ed.), Reviews of Environmental Contamination and Toxicology: Continuation of Residue Reviews. Springer, New York, p.73–145. https://doi.org/10.1007/978-1-4612-2672-7_4
4. Hori Y, Tanaka T, Fujisawa M, et al., 2003. Toxicokinetics of DL-glufosinate enantiomer in human BASTA® poisoning. Biol Pharm Bull, 26(4):540–543. https://doi.org/10.1248/bpb.26.540
5. Kim J, Chun BJ, Moon JM, et al., 2022. Prognostic value of neutrophil to lymphocyte ratio in the diagnosis of neurotoxicity after glufosinate ammonium poisoning. J Toxicol Environ Health Part A, 85(12):511–519. https://doi.org/10.1080/15287394.2022.2040670
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