Affiliation:
1. Department of Medicine, University of Chicago Chicago, Illinois
Abstract
The feeding of a lipid-poor and virtually cholesterol-free residue of mammalian brain mitigates the hypercholesteremia and atherosclerosis ordinarily seen in cholesterol-fed chicks. The probable mechanism depends on the previously demonstrated capacity of oral cerebrosides to convert a large proportion of fecal sterols into unabsorbable coprosterol, thus rendering intestinal cholesterol, unavailable to the body economy.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
Cited by
13 articles.
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