Oxidative Stress by Monoamine Oxidase Mediates Receptor-Independent Cardiomyocyte Apoptosis by Serotonin and Postischemic Myocardial Injury-Reference-Cited by-同舟云学术

Oxidative Stress by Monoamine Oxidase Mediates Receptor-Independent Cardiomyocyte Apoptosis by Serotonin and Postischemic Myocardial Injury

Published:2005-11-22 Issue:21 Volume:112 Page:3297-3305
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Bianchi Pascale¹,Kunduzova Oxana¹,Masini Emanuela¹,Cambon Claudie¹,Bani Daniele¹,Raimondi Laura¹,Seguelas Marie-Helene¹,Nistri Silvia¹,Colucci Wilson¹,Leducq Nathalie¹,Parini Angelo¹

Affiliation:

1. From INSERM U388, Toulouse, France (P.B., O.K., C.C., M.-H.S., A.P.); Department of Preclinical and Clinical Pharmacology, Department of Anatomy, Histology and Forensic Medicine, University of Florence, Florence, Italy (L.M., D.B., L.R., S.N.); Boston University Medical Center, Boston, Mass (W.C.); and Cardiovascular-Thrombosis Research Department, Sanofi-Synthélabo Research, Toulouse, France (N.L.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference32 articles.

1. Disruption of the nonneuronal tph1 gene demonstrates the importance of peripheral serotonin in cardiac function

2. Effect of 5-HT4 receptor stimulation on the pacemaker current If in human isolated atrial myocytes

3. Serotonin is a novel survival factor of cardiomyocytes: mitochondria as a target of 5‐HT2B‐receptor signaling

4. Factors Associated with Progression of Carcinoid Heart Disease

5. Activated platelets contribute to stimulation of cardiac afferents during ischaemia in cats: role of 5‐HT3receptors

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