H 2 S Protects Against Pressure Overload–Induced Heart Failure via Upregulation of Endothelial Nitric Oxide Synthase-Reference-Cited by-同舟云学术

H 2 S Protects Against Pressure Overload–Induced Heart Failure via Upregulation of Endothelial Nitric Oxide Synthase

Published:2013-03-12 Issue:10 Volume:127 Page:1116-1127
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Kondo Kazuhisa¹,Bhushan Shashi¹,King Adrienne L.¹,Prabhu Sumanth D.¹,Hamid Tariq¹,Koenig Steven¹,Murohara Toyoaki¹,Predmore Benjamin L.¹,Gojon Gabriel¹,Gojon Gabriel¹,Wang Rui¹,Karusula Naveena¹,Nicholson Chad K.¹,Calvert John W.¹,Lefer David J.¹

Affiliation:

1. From the Department of Surgery, Division of Cardiothoracic Surgery, Emory University School of Medicine, and The Carlyle Fraser Heart Center, Atlanta, GA (K.K., S.B., A.L.K., B.L.P., N.K., C.K.N., J.WC., D.J.L.); Department of Cardiology, Nagoya University Graduate School of Medicine, Nagoya, Japan (T.M.); Medicinal Chemistry Division, NAN Laboratorios, Monterrey, Mexico (G.G., G.G.); Department of Biology, Lakehead University, Thunder Bay, ON, Canada (R.W.); Division of Cardiovascular Disease,...

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference41 articles.

1. Prognostic Implications of Echocardiographically Determined Left Ventricular Mass in the Framingham Heart Study

2. Regulation of cardiac hypertrophy by intracellular signalling pathways

3. Hydrogen Sulfide and Cell Signaling

4. Hydrogen Sulfide Mediates Cardioprotection Through Nrf2 Signaling

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