Inhibition of S-Adenosylhomocysteine Hydrolase Induces Endothelial Dysfunction via Epigenetic Regulation of p66shc-Mediated Oxidative Stress Pathway-Reference-Cited by-同舟云学术

Inhibition of S-Adenosylhomocysteine Hydrolase Induces Endothelial Dysfunction via Epigenetic Regulation of p66shc-Mediated Oxidative Stress Pathway

Published:2019-05-07 Issue:19 Volume:139 Page:2260-2277
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Xiao Yunjun¹,Xia Junjie¹,Cheng Jinquan¹,Huang Haiyan²,Zhou Yani¹,Yang Xifei²,Su Xuefen³,Ke Yuebin¹,Ling Wenhua⁴

Affiliation:

1. Shenzhen Key Laboratory of Molecular Epidemiology (Y.X., J.X., J.C., Y.Z., YK.), Shenzhen Center for Disease Control and Prevention, Guangdong, China.

2. Key Laboratory of Modern Toxicology of Shenzhen (H.H., X.Y.), Shenzhen Center for Disease Control and Prevention, Guangdong, China.

3. School of Public Health and Primary Care, Faculty of Medicine, The Chinese University of Hong Kong, China (X.S.).

4. Guangdong Provincial Key Laboratory of Food, Nutrition and Health, Department of Nutrition, School of Public Health, Sun Yat-sen University, Guangzhou, Guangdong, China (W.L.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference45 articles.

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4. Plasma S-adenosylhomocysteine is a more sensitive indicator of cardiovascular disease than plasma homocysteine

5. Increased Homocysteine and S-Adenosylhomocysteine Concentrations and DNA Hypomethylation in Vascular Disease

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