Immune Activation Resulting From NKG2D/Ligand Interaction Promotes Atherosclerosis

Author:

Xia Mingcan1,Guerra Nadia1,Sukhova Galina K.1,Yang Kangkang1,Miller Carla K.1,Shi Guo-Ping1,Raulet David H.1,Xiong Na1

Affiliation:

1. From the Center of Molecular Immunology and Infectious Diseases, Departments of Veterinary and Biomedical Sciences (M.X., K.Y., N.X.) and Nutrition (C.K.M.), Pennsylvania State University, University Park; Department of Molecular and Cell Biology, University of California, Berkeley (N.G., D.H.R.); and Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, MA (G.K.S., G.S.). Current address for N.G. is Division of Cell and Molecular Biology, Imperial College, London,...

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference42 articles.

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2. Inflammation, stress, and diabetes

3. Inflammation and metabolic disorders

4. Immunology of atherosclerosis: demonstration of heat shock protein 60 expression and T lymphocytes bearing alpha/beta or gamma/delta receptor in human atherosclerotic lesions;Kleindienst R;Am J Pathol,1993

5. Identification of natural killer cells in human atherosclerotic plaque

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