Constitutive Intracellular Na + Excess in Purkinje Cells Promotes Arrhythmogenesis at Lower Levels of Stress Than Ventricular Myocytes From Mice With Catecholaminergic Polymorphic Ventricular Tachycardia-Reference-Cited by-同舟云学术

Constitutive Intracellular Na + Excess in Purkinje Cells Promotes Arrhythmogenesis at Lower Levels of Stress Than Ventricular Myocytes From Mice With Catecholaminergic Polymorphic Ventricular Tachycardia

Published:2016-06-14 Issue:24 Volume:133 Page:2348-2359
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Willis B. Cicero¹,Pandit Sandeep V.¹,Ponce-Balbuena Daniela¹,Zarzoso Manuel¹,Guerrero-Serna Guadalupe¹,Limbu Bijay¹,Deo Makarand¹,Camors Emmanuel¹,Ramirez Rafael J.¹,Mironov Sergey¹,Herron Todd J.¹,Valdivia Héctor H.¹,Jalife José¹

Affiliation:

1. From University of Michigan, Ann Arbor (B.C.W., S.V.P., D.P.-B., M.Z., G.G.-S., R.J.R., S.M., T.J.H., H.H.V., J.J.); Norfolk State University, VA (B.L., M.D.); University of Tennessee Health Science Center, Memphis (E.C.); and Fundación Centro Nacional de Investigaciones Cardiovasculares, Madrid, Spain (J.J.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference45 articles.

1. Inherited calcium channelopathies in the pathophysiology of arrhythmias

2. A Missense Mutation in a Highly Conserved Region of CASQ2 Is Associated with Autosomal Recessive Catecholamine-Induced Polymorphic Ventricular Tachycardia in Bedouin Families from Israel

3. Absence of triadin, a protein of the calcium release complex, is responsible for cardiac arrhythmia with sudden death in human

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