Dipeptidyl Peptidase-4 Modulates Left Ventricular Dysfunction in Chronic Heart Failure via Angiogenesis-Dependent and -Independent Actions-Reference-Cited by-同舟云学术

Dipeptidyl Peptidase-4 Modulates Left Ventricular Dysfunction in Chronic Heart Failure via Angiogenesis-Dependent and -Independent Actions

Published:2012-10-09 Issue:15 Volume:126 Page:1838-1851
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Shigeta Toshimasa¹,Aoyama Morihiko¹,Bando Yasuko K¹,Monji Akio¹,Mitsui Toko¹,Takatsu Miwa¹,Cheng Xiang-Wu¹,Okumura Takahiro¹,Hirashiki Akihiro¹,Nagata Kohzo¹,Murohara Toyoaki¹

Affiliation:

1. From the Departments of Cardiology (T.S., M.A., Y.K.B., A.M., T.M., X.-W.C., T.O., A.H., T.M.) and Pathophysiology Laboratory Sciences (M.T.), Nagoya University Graduate School of Medicine, and Department of Medical Technology, Nagoya University School of Health Sciences (K.N.), Nagoya, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference38 articles.

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4. The Cysteine-Rich Region of Dipeptidyl Peptidase IV (CD 26) Is the Collagen Binding Site

5. Rat dipeptidyl peptidase IV (DPP IV) exhibits endopeptidase activity with specificity for denatured fibrillar collagens

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