Activation of the Cardiac Proteasome During Pressure Overload Promotes Ventricular Hypertrophy-Reference-Cited by-同舟云学术

Activation of the Cardiac Proteasome During Pressure Overload Promotes Ventricular Hypertrophy

Published:2006-10-24 Issue:17 Volume:114 Page:1821-1828
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Depre Christophe¹,Wang Qian¹,Yan Lin¹,Hedhli Nadia¹,Peter Pallavi¹,Chen Li¹,Hong Chull¹,Hittinger Luc¹,Ghaleh Bijan¹,Sadoshima Junichi¹,Vatner Dorothy E.¹,Vatner Stephen F.¹,Madura Kiran¹

Affiliation:

1. From the Cardiovascular Research Institute (C.D., Q.W., L.Y., N.H., P.P., C.H., L.H., B.G., J.S., D.E.V., S.F.V.), Department of Cell Biology and Molecular Medicine, University of Medicine and Dentistry of New Jersey, Newark, and Department of Biochemistry (L.C., K.M.), Robert Wood Johnson Medical School, University of Medicine and Dentistry of New Jersey, Piscataway.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference37 articles.

1. Increased protein synthesis and degradation in the dog heart during thyroxine administration

2. Biochemical Mechanisms of Cardiac Hypertrophy

3. The Ubiquitin-Proteasome Proteolytic Pathway: Destruction for the Sake of Construction

4. Signalling pathways that mediate skeletal muscle hypertrophy and atrophy

5. Atrophic Remodeling of the Heart In Vivo Simultaneously Activates Pathways of Protein Synthesis and Degradation

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