ROS‐Mediated PARP Activity Undermines Mitochondrial Function After Permeability Transition Pore Opening During Myocardial Ischemia

ROS‐Mediated PARP Activity Undermines Mitochondrial Function After Permeability Transition Pore Opening During Myocardial Ischemia–Reperfusion

Published:2013-03-12 Issue:2 Volume:2 Page:
ISSN:2047-9980
Container-title:Journal of the American Heart Association
language:en
Short-container-title:JAHA

Author:

Schriewer Jacqueline M.¹,Peek Clara Bien²,Bass Joseph²,Schumacker Paul T.¹

Affiliation:

1. Division of Neonatology, Department of Pediatrics, Northwestern University Feinberg School of Medicine, Chicago, IL

2. Division of Endocrinology, Metabolism, and Molecular Medicine, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, IL

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference61 articles.

1. Heart Disease and Stroke Statistics—2012 Update

2. Myocardial Reperfusion Injury

3. Ischemia-Reperfusion-Induced Cardiac Injury

4. Cell death during ischemia: relationship to mitochondrial depolarization and ROS generation

5. Overexpression of MnSOD Protects Against Myocardial Ischemia/reperfusion Injury in Transgenic Mice

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