Inducible Overexpression of GLUT1 Prevents Mitochondrial Dysfunction and Attenuates Structural Remodeling in Pressure Overload but Does Not Prevent Left Ventricular Dysfunction-Reference-Cited by-同舟云学术

Inducible Overexpression of GLUT1 Prevents Mitochondrial Dysfunction and Attenuates Structural Remodeling in Pressure Overload but Does Not Prevent Left Ventricular Dysfunction

Published:2013-09-26 Issue:5 Volume:2 Page:
ISSN:2047-9980
Container-title:Journal of the American Heart Association
language:en
Short-container-title:JAHA

Author:

Pereira Renata O.¹,Wende Adam R.¹,Olsen Curtis¹,Soto Jamie¹,Rawlings Tenley¹,Zhu Yi¹,Anderson Steven M.²,Dale Abel E.¹

Affiliation:

1. Division of Endocrinology, Metabolism and Diabetes, and Program in Molecular Medicine, University of Utah School of Medicine, Salt Lake City, UT

2. Department of Pathology, University of Colorado School of Medicine, Anshutz Medical Campus, Aurora, CO

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference49 articles.

1. Mitochondrial energy metabolism in heart failure: a question of balance

2. Energy metabolism of the heart: From basic concepts to clinical applications applications

3. Glucose transport in the heart

4. Persistent Myocardial Ischemia Increases GLUT1 Glucose Transporter Expression in Both Ischemic and Non-ischemic Heart Regions

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