Junctophilin-2 Expression Silencing Causes Cardiocyte Hypertrophy and Abnormal Intracellular Calcium-Handling-Reference-Cited by-同舟云学术

Junctophilin-2 Expression Silencing Causes Cardiocyte Hypertrophy and Abnormal Intracellular Calcium-Handling

Published:2011-03 Issue:2 Volume:4 Page:214-223
ISSN:1941-3289
Container-title:Circulation: Heart Failure
language:en
Short-container-title:Circ Heart Fail

Author:

Landstrom Andrew P.¹,Kellen Cherisse A.¹,Dixit Sayali S.¹,van Oort Ralph J.¹,Garbino Alejandro¹,Weisleder Noah¹,Ma Jianjie¹,Wehrens Xander H.T.¹,Ackerman Michael J.¹

Affiliation:

1. From the Department of Molecular Pharmacology & Experimental Therapeutics (A.L., C.K., M.A.), Windland Smith Rice Sudden Death Genomics Laboratory, Mayo Clinic, Rochester, MN; Department of Molecular Physiology and Biophysics, and Medicine (in Cardiology) (S.D., R.O., A.G., X.W.), Baylor College of Medicine, Houston, TX; Department of Physiology and Biophysics (N.W., J.M.), Robert Wood Johnson Medical School, Piscataway, NJ; Departments of Medicine and Pediatrics, Divisions of Cardiovascular...

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference49 articles.

1. Characterization of Human Junctophilin Subtype Genes

2. Molecular evolution of the junctophilin gene family

3. Hypertension-induced remodeling of cardiac excitation-contraction coupling in ventricular myocytes occurs prior to hypertrophy development

4. Orphaned ryanodine receptors in the failing heart

5. Effect of Metabolic Inhibition on Couplon Behavior in Rabbit Ventricular Myocytes

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