Upregulation of Nox4 by Hypertrophic Stimuli Promotes Apoptosis and Mitochondrial Dysfunction in Cardiac Myocytes-Reference-Cited by-同舟云学术

Upregulation of Nox4 by Hypertrophic Stimuli Promotes Apoptosis and Mitochondrial Dysfunction in Cardiac Myocytes

Published:2010-04-16 Issue:7 Volume:106 Page:1253-1264
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Ago Tetsuro¹,Kuroda Junya¹,Pain Jayashree¹,Fu Cexiong¹,Li Hong¹,Sadoshima Junichi¹

Affiliation:

1. From the Department of Cell Biology and Molecular Medicine (T.A., J.K., J.P., J.S.), Cardiovascular Research Institute; and Center for Advanced Proteomics Research (C.F., H.L.), Department of Biochemistry and Molecular Biology, University of Medicine and Dentistry of New Jersey, New Jersey Medical School, Newark.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference33 articles.

1. Redox-Dependent Transcriptional Regulation

2. Reactive Oxygen Species, Mitochondria, and NAD(P)H Oxidases in the Development and Progression of Heart Failure

3. Mitochondrial oxidative stress and dysfunction in myocardial remodelling

4. Mitochondria, Oxidants, and Aging

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