Ca 2+ -Independent Alterations in Diastolic Sarcomere Length and Relaxation Kinetics in a Mouse Model of Lipotoxic Diabetic Cardiomyopathy-Reference-Cited by-同舟云学术

Ca 2+ -Independent Alterations in Diastolic Sarcomere Length and Relaxation Kinetics in a Mouse Model of Lipotoxic Diabetic Cardiomyopathy

Published:2009-01-02 Issue:1 Volume:104 Page:95-103
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Flagg Thomas P.¹,Cazorla Olivier¹,Remedi Maria S.¹,Haim Todd E.¹,Tones Michael A.¹,Bahinski Anthony¹,Numann Randal E.¹,Kovacs Attila¹,Schaffer Jean E.¹,Nichols Colin G.¹,Nerbonne Jeanne M.¹

Affiliation:

1. From the Departments of Cell Biology and Physiology (T.P.F., M.S.R., C.G.N.), Molecular Biology and Pharmacology (J.M.N.), and Internal Medicine (A.K., J.E.S.), Washington University School of Medicine, St Louis, Mo; Institut National de la Santé et de la Recherche Médicale, U-637 (O.C.), Université Montpellier 1, Unité de Formation et de Recherche de Médecine, Montpellier, France; and Pfizer Global Research & Development (T.E.H., M.A.T., A.B., R.E.N.), Chesterfield, Mo.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference39 articles.

1. Metabolic Mechanisms in Heart Failure

2. Diabetic Cardiomyopathy Revisited

3. Regulation of energy substrate metabolism in the diabetic heart

4. Diastolic dysfunction is associatedwith altered myocardial metabolism inasymptomatic normotensive patientswith well-controlled type 2 diabetes mellitus

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