Phosphodiesterase 5: A Novel Therapeutic Target in Long QT Syndrome
Author:
Affiliation:
1. Dipartimento di Biotecnologie e Bioscienze, Università degli Studi di Milano-Bicocca, Milan, Italy.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
Link
https://www.ahajournals.org/doi/pdf/10.1161/CIRCRESAHA.121.319851
Reference15 articles.
1. PDE5 Inhibition Suppresses Ventricular Arrhythmias by Reducing SR Ca 2+ Content
2. cGMP Catabolism by Phosphodiesterase 5A Regulates Cardiac Adrenergic Stimulation by NOS3-Dependent Mechanism
3. Compartmentalization of Cardiac β-Adrenergic Inotropy Modulation by Phosphodiesterase Type 5
4. Action potential duration determines sarcoplasmic reticulum Ca2+reloading in mammalian ventricular myocytes
5. Action Potential Prolongation, β-Adrenergic Stimulation, and Angiotensin II as Co-factors in Sarcoplasmic Reticulum Instability
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