Matrix Metalloproteinase-2 Plays a Critical Role in the Pathogenesis of White Matter Lesions After Chronic Cerebral Hypoperfusion in Rodents-Reference-Cited by-同舟云学术

Matrix Metalloproteinase-2 Plays a Critical Role in the Pathogenesis of White Matter Lesions After Chronic Cerebral Hypoperfusion in Rodents

Published:2006-11 Issue:11 Volume:37 Page:2816-2823
ISSN:0039-2499
Container-title:Stroke
language:en
Short-container-title:Stroke

Author:

Nakaji Kayoko¹,Ihara Masafumi¹,Takahashi Chiaki¹,Itohara Shigeyoshi¹,Noda Makoto¹,Takahashi Ryosuke¹,Tomimoto Hidekazu¹

Affiliation:

1. From the Department of Neurology (K.N., M.I., R.T., H.T.), Horizontal Medical Research Organization (M.I.), the Department of Molecular Oncology (M.N.), and The 21st Century Center of Excellence Program, Department of Oncology (C.T.), Kyoto University Graduate School of Medicine, Kyoto, Japan; and the Laboratory for Behavioral Genetics (S.I.), RIKEN Brain Science Institute, Wako, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Advanced and Specialized Nursing,Cardiology and Cardiovascular Medicine,Neurology (clinical)

Reference23 articles.

1. Cerebral circulation and oxygen metabolism associated with subclinical periventricular hyperintensity as shown by magnetic resonance imaging

2. Glial activation and white matter changes in the rat brain induced by chronic cerebral hypoperfusion: an immunohistochemical study

3. White Matter Lesions and Glial Activation in a Novel Mouse Model of Chronic Cerebral Hypoperfusion

4. How Matrix Metalloproteinases Regulate Cell Behavior

5. Increased gelatinase A (MMP-2) and gelatinase B (MMP-9) activities in human brain after focal ischemia

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