Anti-Inflammatory Effects of Nicotinic Acid in Human Monocytes Are Mediated by GPR109A Dependent Mechanisms-Reference-Cited by-同舟云学术

Anti-Inflammatory Effects of Nicotinic Acid in Human Monocytes Are Mediated by GPR109A Dependent Mechanisms

Published:2012-03 Issue:3 Volume:32 Page:669-676
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:Arterioscler Thromb Vasc Biol.

Author:

Digby Janet E.¹,Martinez Fernando¹,Jefferson Andrew¹,Ruparelia Neil¹,Chai Joshua¹,Wamil Malgorzata¹,Greaves David R.¹,Choudhury Robin P.¹

Affiliation:

1. From the Department of Cardiovascular Medicine (J.E.D., A.J., J.C., M.W., N.R., R.P.C.) and Sir William Dunn School of Pathology (F.M., D.R.G.), University of Oxford, Oxford, United Kingdom.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference42 articles.

1. Fifteen year mortality in Coronary Drug Project patients: Long-term benefit with niacin

2. Effects of High-Dose Modified-Release Nicotinic Acid on Atherosclerosis and Vascular Function

3. Extended-Release Niacin or Ezetimibe and Carotid Intima–Media Thickness

4. Niacin stimulates adiponectin secretion through the GPR109A receptor

5. PUMA-G and HM74 are receptors for nicotinic acid and mediate its anti-lipolytic effect

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