APOC3 Loss-of-Function Mutations, Remnant Cholesterol, Low-Density Lipoprotein Cholesterol, and Cardiovascular Risk-Reference-Cited by-同舟云学术

APOC3 Loss-of-Function Mutations, Remnant Cholesterol, Low-Density Lipoprotein Cholesterol, and Cardiovascular Risk

Published:2018-03 Issue:3 Volume:38 Page:660-668
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:Arterioscler Thromb Vasc Biol.

Author:

Wulff Anders B.¹,Nordestgaard Børge G.¹,Tybjærg-Hansen Anne¹

Affiliation:

1. From the Department of Clinical Biochemistry, Rigshospitalet (A.B.W., A.T.-H.), Department of Clinical Biochemistry, Herlev and Gentofte Hospital (B.G.N.), and Copenhagen City Heart Study, Frederiksberg Hospital (B.G.N., A.T.-H.), Copenhagen University Hospital, University of Copenhagen, Denmark; Department of Clinical Biochemistry, Zealand University Hospital, Denmark (A.B.W.); and Copenhagen General Population Study, Herlev and Gentofte Hospital, Denmark (B.G.N., A.T.-H.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference32 articles.

1. Loss-of-Function Mutations inAPOC3and Risk of Ischemic Vascular Disease

2. Loss-of-Function Mutations in APOC3, Triglycerides, and Coronary Disease

3. Antisense Inhibition of Apolipoprotein C-III in Patients with Hypertriglyceridemia

4. Targeting APOC3 in the Familial Chylomicronemia Syndrome

5. Genetically elevated non-fasting triglycerides and calculated remnant cholesterol as causal risk factors for myocardial infarction

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