Therapeutic Silencing of MicroRNA-33 Inhibits the Progression of Atherosclerosis in Ldlr −/− Mice

Therapeutic Silencing of MicroRNA-33 Inhibits the Progression of Atherosclerosis in Ldlr −/− Mice—Brief Report

Published:2013-08 Issue:8 Volume:33 Page:1973-1977
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:Arterioscler Thromb Vasc Biol.

Author:

Rotllan Noemi¹,Ramírez Cristina M.¹,Aryal Binod¹,Esau Christine C.¹,Fernández-Hernando Carlos¹

Affiliation:

1. From the Department of Medicine, Leon H. Charney Division of Cardiology and Cell Biology and the Marc and Ruti Bell Vascular Biology and Disease Program, New York University School of Medicine, New York, NY (N.R., C.M.R., B.A., C.F.-H.); and Regulus Therapeutics, San Diego, CA (C.C.E.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference10 articles.

1. Expression of miR-33 from an SREBP2 Intron Inhibits Cholesterol Export and Fatty Acid Oxidation*

2. MicroRNA-33 encoded by an intron of sterol regulatory element-binding protein 2 (Srebp2) regulates HDL in vivo

3. miR-33 links SREBP-2 induction to repression of sterol transporters

4. MicroRNA-33 and the SREBP Host Genes Cooperate to Control Cholesterol Homeostasis

5. MiR-33 Contributes to the Regulation of Cholesterol Homeostasis

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