Loss of SR-A and CD36 Activity Reduces Atherosclerotic Lesion Complexity Without Abrogating Foam Cell Formation in Hyperlipidemic Mice

Author:

Manning-Tobin Jennifer J.1,Moore Kathryn J.1,Seimon Tracie A.1,Bell Susan A.1,Sharuk Maia1,Alvarez-Leite Jacqueline I.1,de Winther Menno P.J.1,Tabas Ira1,Freeman Mason W.1

Affiliation:

1. From the Lipid Metabolism Unit (J.J.M.-T., K.J.M., S.A.B., M.S., J.I.A.-L., M.W.F.) and the Center for Computational & Integrative Biology (M.W.F.), Massachusetts General Hospital, Harvard Medical School, Boston; the Department of Medicine (T.A.S., I.T.) and the Departments of Pathology & Cell Biology and Physiology & Cellular Biophysics (I.T.), Columbia University, New York; the Federal University of Minas Gerais (J.I.A.-L.), Belo Horizonte, Brazil; and the Department of Molecular...

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

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