Dachsous1–Fat4 Signaling Controls Endothelial Cell Polarization During Lymphatic Valve Morphogenesis

Dachsous1–Fat4 Signaling Controls Endothelial Cell Polarization During Lymphatic Valve Morphogenesis—Brief Report

Published:2017-09 Issue:9 Volume:37 Page:1732-1735
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:Arterioscler Thromb Vasc Biol.

Author:

Pujol Francoise¹,Hodgson Tina¹,Martinez-Corral Ines¹,Prats Anne-Catherine¹,Devenport Danelle¹,Takeichi Masatoshi¹,Genot Elisabeth¹,Mäkinen Taija¹,Francis-West Philippa¹,Garmy-Susini Barbara¹,Tatin Florence¹

Affiliation:

1. From the I2MC INSERM UMR 1048, Toulouse Cedex, France (F.P., A.-C.P., B.G.-S., F.T.); Department Craniofacial Development and Stem Cell Biology, King’s College London, United Kingdom (T.H., P.F.-W.); Rudbeck Laboratory, Department Immunology, Genetics and Pathology, Uppsala University, Sweden (I.M.-C., T.M.); Department of Molecular Biology, Princeton University, NJ (D.D.); Laboratory for Cell Adhesion and Tissue Patterning, RIKEN Center for Developmental Biology, Kobe, Japan (M.T.); and INSERM,...

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference13 articles.

1. Van Maldergem syndrome: further characterisation and evidence for neuronal migration abnormalities and autosomal recessive inheritance

2. Mutations in genes encoding the cadherin receptor-ligand pair DCHS1 and FAT4 disrupt cerebral cortical development

3. Hennekam syndrome can be caused by FAT4 mutations and be allelic to Van Maldergem syndrome

4. Frizzled/PCP signalling: a conserved mechanism regulating cell polarity and directed motility

5. Loss of Fat4 disrupts PCP signaling and oriented cell division and leads to cystic kidney disease

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