Laminar Shear Stress Inhibits Endothelial Cell Metabolism via KLF2-Mediated Repression of PFKFB3-Reference-Cited by-同舟云学术

Laminar Shear Stress Inhibits Endothelial Cell Metabolism via KLF2-Mediated Repression of PFKFB3

Published:2015-01 Issue:1 Volume:35 Page:137-145
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:Arterioscler Thromb Vasc Biol.

Author:

Doddaballapur Anuradha¹,Michalik Katharina M.¹,Manavski Yosif¹,Lucas Tina¹,Houtkooper Riekelt H.¹,You Xintian¹,Chen Wei¹,Zeiher Andreas M.¹,Potente Michael¹,Dimmeler Stefanie¹,Boon Reinier A.¹

Affiliation:

1. From the Institute for Cardiovascular Regeneration, Center of Molecular Medicine, Goethe University, Frankfurt am Main, Germany (A.D., K.M.M., Y.M., T.L., S.D., R.A.B.); The Laboratory Genetic Metabolic Diseases, Academic Medical Center, Amsterdam, The Netherlands (R.H.H.); The Max-Delbrück-Center, Berlin, Germany (X.Y., W.C.); Department of Cardiology, Internal Medicine III, Goethe University Hospital Frankfurt, Frankfurt am Main, Germany (A.M.Z.); Angiogenesis and Metabolism Laboratory, Max Planck...

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference19 articles.

1. Heterozygous Deficiency of PHD2 Restores Tumor Oxygenation and Inhibits Metastasis via Endothelial Normalization

2. Basic and Therapeutic Aspects of Angiogenesis

3. Role of histone deacetylases in transcription factor regulation and cell cycle modulation in endothelial cells in response to disturbed flow

4. KLF2 provokes a gene expression pattern that establishes functional quiescent differentiation of the endothelium

5. Integration of flow-dependent endothelial phenotypes by Kruppel-like factor 2

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