Matrix Metalloproteinase 2 and 9 Dysfunction Underlie Vascular Stiffness in Circadian Clock Mutant Mice-Reference-Cited by-同舟云学术

Matrix Metalloproteinase 2 and 9 Dysfunction Underlie Vascular Stiffness in Circadian Clock Mutant Mice

Published:2010-12 Issue:12 Volume:30 Page:2535-2543
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Anea Ciprian B.¹,Ali M. Irfan¹,Osmond Jessica M.¹,Sullivan Jennifer C.¹,Stepp David W.¹,Merloiu Ana M.¹,Rudic R. Daniel¹

Affiliation:

1. From the Department of Pharmacology and Toxicology (C.B.A., A.M.M., and R.D.R.), Medical College of Georgia, Augusta; the Vascular Biology Center (M.I.A., J.M.O., J.C.S., and D.W.S.), Medical College of Georgia, Augusta; and the Department of Physiology (D.W.S.), Medical College of Georgia, Augusta.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference35 articles.

1. Vascular Disease in Mice With a Dysfunctional Circadian Clock

2. Mutation of the Circadian Clock Gene Per2 Alters Vascular Endothelial Function

3. Increased Vascular Senescence and Impaired Endothelial Progenitor Cell Function Mediated by Mutation of Circadian Gene Per2

4. Genetic Components of the Circadian Clock Regulate Thrombogenesis In Vivo

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