Hyperoxia Causes Mitochondrial Fragmentation in Pulmonary Endothelial Cells by Increasing Expression of Pro-Fission Proteins-Reference-Cited by-同舟云学术

Hyperoxia Causes Mitochondrial Fragmentation in Pulmonary Endothelial Cells by Increasing Expression of Pro-Fission Proteins

Published:2018-03 Issue:3 Volume:38 Page:622-635
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:Arterioscler Thromb Vasc Biol.

Author:

Ma Cui¹,Beyer Andreas M.¹,Durand Matthew¹,Clough Anne V.¹,Zhu Daling¹,Norwood Toro Laura¹,Terashvili Maia¹,Ebben Johnathan D.¹,Hill R. Blake¹,Audi Said H.¹,Medhora Meetha¹,Jacobs Elizabeth R.¹

Affiliation:

1. From the College of Medical Laboratory Science and Technology, Harbin Medical University, Daqing, China (C.M., D.Z., M.M., E.R.J.); Department of Medicine (C.M., A.M.B., A.C., L.N., J.E., M.M., E.R.J.), Department of Physical Medicine and Rehabilitation (M.D.), Department of Physiology (A.M.B., M.M., E.R.J.), Department of Biochemistry (B.H.), Department of Radiation Oncology (M.M.), Department of Biophysics (N.H.), and Cardiovascular Center (M.T., C.M., A.B., M.D., M.M., E.R.J.), Medical College of...

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference48 articles.

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4. In vivo detection of hyperoxia-induced pulmonary endothelial cell death using 99mTc-Duramycin

5. Mitochondrial fission and fusion in human diseases.;Archer SL;N Engl J Med,2014

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