TRPV4 (Transient Receptor Potential Vanilloid 4) Channel–Dependent Negative Feedback Mechanism Regulates G q Protein–Coupled Receptor–Induced Vasoconstriction-Reference-Cited by-同舟云学术

TRPV4 (Transient Receptor Potential Vanilloid 4) Channel–Dependent Negative Feedback Mechanism Regulates G q Protein–Coupled Receptor–Induced Vasoconstriction

Published:2018-03 Issue:3 Volume:38 Page:542-554
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:Arterioscler Thromb Vasc Biol.

Author:

Hong Kwangseok¹,Cope Eric L.¹,DeLalio Leon J.¹,Marziano Corina¹,Isakson Brant E.¹,Sonkusare Swapnil K.¹

Affiliation:

1. From the Robert M. Berne Cardiovascular Research Center (K.H., E.L.C., L.J.D., C.M., B.E.I., S.K.S.), Department of Molecular Physiology and Biological Physics (C.M., B.E.I., S.K.S.), and Department of Pharmacology (L.J.D., S.K.S), University of Virginia-School of Medicine, Charlottesville.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference64 articles.

1. Cat cerebral arterial smooth muscle cells express cytochrome P450 4A2 enzyme and produce the vasoconstrictor 20-HETE which enhances L-type Ca2+current

2. Mechanical activation of angiotensin II type 1 receptors causes actin remodelling and myogenic responsiveness in skeletal muscle arterioles

3. Gq-coupled receptors as mechanosensors mediating myogenic vasoconstriction

4. The angiotensin II receptor type 1b is the primary sensor of intraluminal pressure in cerebral artery smooth muscle cells

5. Stretch–Activation of Angiotensin II Type 1 a Receptors Contributes to the Myogenic Response of Mouse Mesenteric and Renal Arteries

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