SMIT1 Expression in Arterial Tissue: A Potential New Trigger of Vascular Dysfunctions and ROS Production in Rats-Reference-Cited by-同舟云学术

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SMIT1 Expression in Arterial Tissue: A Potential New Trigger of Vascular Dysfunctions and ROS Production in Rats

Published:2024-05 Issue:5 Volume:44 Page:
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Grandperrin Antoine¹,Strock Eva¹,Petit Léna¹^ORCID,Risdon Sydney¹,Boulghobra Doria¹,Gayrard Sandrine¹^ORCID,Reboul Cyril¹^ORCID,Walther Guillaume¹^ORCID,Battault Sylvain¹^ORCID,Meyer Grégory¹

Affiliation:

1. UPR-4278, LaPEC, Laboratory of Cardiovascular Experimental Physiology, Avignon University, France.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Reference5 articles.

1. Effects of Sugar-Sweetened Beverage Consumption on Microvascular and Macrovascular Function in a Healthy Population

2. Sodium-myoinositol cotransporter-1, SMIT1, mediates the production of reactive oxygen species induced by hyperglycemia in the heart

3. Inositol transport proteins

4. Molecular physiology of sodium-glucose cotransporters.

5. Angiotensin II-induced upregulation of SGLT1 and 2 contributes to human microparticle‐stimulated endothelial senescence and dysfunction: protective effect of gliflozins

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