Increased Inhibition of Inward Rectifier K + Channels by Angiotensin II in Small-Diameter Coronary Artery of Isoproterenol-Induced Hypertrophied Model-Reference-Cited by-同舟云学术

Increased Inhibition of Inward Rectifier K + Channels by Angiotensin II in Small-Diameter Coronary Artery of Isoproterenol-Induced Hypertrophied Model

Published:2007-08 Issue:8 Volume:27 Page:1768-1775
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Park Won Sun¹,Ko Jae-Hong¹,Kim Nari¹,Son Youn Kyoung¹,Kang Sung Hyun¹,Warda Mohamad¹,Jung In Duk¹,Park Yeong-Min¹,Han Jin¹

Affiliation:

1. From the Mitochondrial Signaling Laboratory (W.S.P., J.-H.K., N.K., Y.K.S., S.H.K., M.W., J.H.), Mitochondria Research Group, Department of Physiology and Biophysics, College of Medicine, Biohealth Products Research Center, Cardiovascular and Metabolic Disease Research Center, Inje University, Busan, Korea; and the Department of Microbiology and Immunology and National Research Laboratory of Dendritic Cell Differentiation & Regulation (I.D.J., Y.-M.P.), Medical Research Institute, Pusan National...

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference50 articles.

1. Susceptibility of hypertrophied rat hearts to ventricular fibrillation during acute ischemia†

2. Mechanisms of ventricular arrhythmias in cardiac failure and hypertrophy

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