Increased Secretion of Lipoproteins in Transgenic Mice Expressing Human D374Y PCSK9 Under Physiological Genetic Control-Reference-Cited by-同舟云学术

Increased Secretion of Lipoproteins in Transgenic Mice Expressing Human D374Y PCSK9 Under Physiological Genetic Control

Published:2010-07 Issue:7 Volume:30 Page:1333-1339
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Herbert Bronwen¹,Patel Dilipkumar¹,Waddington Simon N.¹,Eden Emily R.¹,McAleenan Alexandra¹,Sun Xi-Ming¹,Soutar Anne K.¹

Affiliation:

1. From the Medical Research Council Clinical Sciences Centre (B.H., D.P., E.R.E., A.M., X.-M.S., and A.K.S.), Imperial College London, London, England; and the Institute for Women’s Health (S.N.W.), University College London. Dr Eden is now with the UCL Institute of Ophthalmology, University College London.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference27 articles.

1. Mutations in PCSK9 cause autosomal dominant hypercholesterolemia

2. An orchestrated gene expression component of neuronal programmed cell death revealed by cDNA array analysis

3. Apolipoprotein B100 Metabolism in Autosomal-Dominant Hypercholesterolemia Related to Mutations inPCSK9

4. Novel putative SREBP and LXR target genes identified by microarray analysis in liver of cholesterol-fed mice

5. Combined analysis of oligonucleotide microarray data from transgenic and knockout mice identifies direct SREBP target genes

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