Deletion of Fstl1 (Follistatin-Like 1) From the Endocardial/Endothelial Lineage Causes Mitral Valve Disease-Reference-Cited by-同舟云学术

Deletion of Fstl1 (Follistatin-Like 1) From the Endocardial/Endothelial Lineage Causes Mitral Valve Disease

Published:2017-09 Issue:9 Volume:37 Page:
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:Arterioscler Thromb Vasc Biol.

Author:

Prakash Stuti¹,Borreguero Luis J.J.¹,Sylva Marc¹,Flores Ruiz Lorena¹,Rezai Fereshte¹,Gunst Quinn D.¹,de la Pompa José-Luis¹,Ruijter Jan M.¹,van den Hoff Maurice J.B.¹

Affiliation:

1. From the Department of Medical Biology, Academic Medical Center, Amsterdam, The Netherlands (S.P., M.S., F.R., Q.D.G., J.M.R., M.J.B.v.d.H.); Cardiovascular Imaging Laboratory, Centro Nacional de Investigación Cardiovascular, Madrid, Spain (L.J.J.B., L.F.R.); and Intercellular Signaling in Cardiovascular Development and Disease Laboratory, Centro Nacional de Investigación Cardiovascular, Madrid, Spain (J.-L.d.l.P.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference61 articles.

1. Another Chromosomal Locus for Mitral Valve Prolapse

2. Genetics of Valvular Heart Disease

3. Lineage and Morphogenetic Analysis of the Cardiac Valves

4. Epicardially derived fibroblasts preferentially contribute to the parietal leaflets of the atrioventricular valves in the murine heart

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