Smooth Muscle–Targeted Knockout of Connexin43 Enhances Neointimal Formation in Response to Vascular Injury-Reference-Cited by-同舟云学术

Smooth Muscle–Targeted Knockout of Connexin43 Enhances Neointimal Formation in Response to Vascular Injury

Published:2007-05 Issue:5 Volume:27 Page:1037-1042
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Liao Yongbo¹,Regan Christopher P.¹,Manabe Ichiro¹,Owens Gary K.¹,Day Kathy H.¹,Damon Dave N.¹,Duling Brian R.¹

Affiliation:

1. From the Department of Anesthesiology (Y.L.), Department of Molecular Physiology and Biological Physics (G.K.O., D.N.D., B.R.D.), and Cardiovascular Research Center (K.H.D.), University of Virginia, Charlottesville, Va; Department of Pharmacology (C.P.R.), Merck Research Laboratories, West Point, Pa; Department of Cardiology (I.M.), University of Tokyo, Tokyo, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference48 articles.

1. Connexin Genes and Cell Growth Control

2. Gap junctions: New tools, new answers, new questions

3. Elevation of intracellular calcium in smooth muscle causes endothelial cell generation of NO in arterioles

4. Central Role of Connexin40 in the Propagation of Electrically Activated Vasodilation in Mouse Cremasteric Arterioles In Vivo

5. Gap junctions: structure and function (Review)

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