C-Reactive Protein-Mediated Vascular Injury Requires Complement

Author:

Hage Fadi G.1,Oparil Suzanne1,Xing Dongqi1,Chen Yiu-Fai1,McCrory Mark A.1,Szalai Alexander J.1

Affiliation:

1. From the Vascular Biology and Hypertension Program, Division of Cardiovascular Disease (F.G.H., S.O., D.X., F.H., Y.-F.C.) and the Division of Clinical Immunology and Rheumatology (M.A.M., A.J.S.), Department of Medicine, The University of Alabama at Birmingham, and Section of Cardiology (F.G.H.), Birmingham Veteran’s Administration Medical Center, Birmingham, Ala.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference46 articles.

1. Exaggerated Neointima Formation in Human C-Reactive Protein Transgenic Mice Is IgG Fc Receptor Type I (FcγRI)-Dependent

2. Estrogen Treatment Abrogates Neointima Formation in Human C-Reactive Protein Transgenic Mice

3. C-Reactive Protein Binding to Murine Leukocytes Requires Fcγ Receptors

4. Structural recognition and functional activation of FcγR by innate pentraxins

5. Hage FG McCrory MA Szalai AJ. C-reactive protein and cardiovascular disease: Lessons learned from studying genetically engineered mice. In: Nagasawa S ed. C-Reactive Protein–New Research. Hauppauge NY: Nova Publishers; 2008: 83–116.

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